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An Urban Experience
L. Findji1
1Fitzpatrick Referrals, Oncology and Soft Tissue surgery, Guildford, United Kingdom
Laryngeal paralysis (LP) is a common cause of upper airway obstruction resulting from a dysfunction of
the recurrent laryngeal nerve(s) resulting in paresis progressing to paralysis of all intrinsic laryngeal muscles except for the cricothyroid muscle. It is most frequently an acquired disease affecting older dogs of large breeds, being an early sign of a general polyneuropathy.
The diagnosis of LP is essentially clinical. Early clinical signs include a change in phonation, inspiratory
stridor and exercise intolerance. Gagging, coughing and retching, as well as vomiting/regurgitations and dysphagia may also be reported. Later in the course of the disease, dyspnoea, respiratory distress, cyanosis and syncopes may be observed. A suspected LP
can be con rmed in several ways. Although other diagnostic tests have been reported, the most common is direct or videoscopic transoral observation of the larynx under light general anaesthesia. Diagnostic
tests aiming at screening for concurrent diseases are also carried out: haematology, blood biochemistry, thyroid function exploration and acetylcholine receptor antibody titres (myasthenia gravis). Thoracic x-rays are also recommended to rule out pre-existing aspiration pneumonia and other concurrent diseases which could affect the prognosis, such as megaoesophagus.
The larynx mainly consists of 4 cartilages: the epiglottic, arytenoid (paired), thyroid and cricoid cartilages. These cartilages are united by joints ( brous or synovial) and muscles. The combined actions of intrinsic laryngeal muscles on these cartilages modify the size, shape and position of the glottis, rostral opening to the (infraglottic) laryngeal lumen and trachea. All intrinsic laryngeal muscles except the cricothyroid, which is innervated
by the cranial laryngeal nerve, are innervated by the caudal laryngeal nerve, terminal segment of the recurrent laryngeal nerve (RLN). The RLN originates in the thorax from the vagus nerve and courses cranially up to the larynx, dorsolaterally to the trachea.
Currently, only two types of techniques are commonly used for de nitive surgical correction of LP: arytenoid lateralisation techniques and partial laryngectomy techniques. Unilateral arytenoid lateralisation (UAL) is the most commonly used technique1, as it appeared associated with fewer complications than bilateral arytenoid lateralisation and partial laryngectomy2. It has the author’s preference and will be the only treatment described here.
For a lateral approach, the animal is placed in lateral recumbency and a cushion can be placed under its neck to elevate the laryngeal area towards the surgeon. The skin is incised over the larynx, ventrally and parallel to
the external jugular vein. The subcutaneous connective tissues and cutaneous muscles overlying the larynx are re ected by a combination of blunt and sharp dissection. The thyropharyngeus and cricopharyngeus muscles are then visible and the dorsal border of the thyroid cartilage lamina is palpable through them. Rather than transecting the thyropharyngeus muscle transversely, we prefer to separate its  bres by blunt dissection over the middle of the dorsal edge of the thyroid cartilage. The separated  bres are then retracted with blunt Gelpi retractors, which provides enough exposure of the thyroid cartilage lamina, covered by a fascial membrane. This laryngeal fascial membrane is incised and the laryngeal mucosa
is detached from the medial aspect of the lamina of the thyroid cartilage. This is done by combination of scalpel incision over the dorsal edge of the thyroid cartilage
and blunt dissection with a Freer periosteal elevator or cotton-buds. Dissection is performed delicately to avoid perforation of the laryngeal mucosa and penetration into the lumen of the larynx. In addition, dissection is limited cranially to avoid damage to the cranial laryngeal nerve, which carries  bres supplying the sensory information from laryngeal mucosa. Preserving these  bres may minimise the incidence of postoperative aspiration of food. The lamina of the thyroid cartilage is retracted laterally using one stay suture. Disarticulation of the cricothyroid joint is not necessary for suf cient exposure of the arytenoid cartilage. The muscular process of the arytenoid is palpated and the cricoarytenoideus dorsalis is transected just caudally to it. The caudal part of the cricoarytenoid joint capsule is incised, but its rostral part is left intact to limit the later abduction of the arytenoid. The author does not section the interarytenoid structures (interarytenoid “band”) in order to minimise the laryngeal disruption and decrease the risk of mucosal perforation. One or two “simple interrupted” sutures are placed between the dorsolateral aspect of the cricoid cartilage and the muscular process of the arytenoid cartilage (Figure 1). Placement of the suture through the cricoid cartilage is done in a caudo-rostral direction. The caudal border of the cricoid cartilage is only palpated: dissection

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